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/27 Date : 00.00.00

Name of the Patient : Abc Xyzpa B. Poolmn / M / 54 yrs.
Referred by : Dr. Abc Xyzbar.
Examination : Intracranial and Neck M.R.A.


C/O giddiness since 1 month.
H/O right sided hemiparesis with loss of speech since 00.00.00.
Known hypertensive/diabetic.


The brain was screened with 5 mm thick T2 Weighted axial images.

Intracranial and neck MRA were performed with 3D TOF and 2D TOF sequences, respectively.


There is evidence of a diffuse area of hyperintensity on the T2 Weighted images within the left fronto-temporal lobes. Areas of hyperintensity on the T1 Weighted images suggestive of subacute blood are seen within this lesion. This lesion would represent a hemorrhagic infarct.

Small hyperintense areas on the T2 Weighted images are noted within the right lentiform nucleus and left paraatrial white matter and these are most likely ischemic in etiology.

A lacunar infarct is seen in the pons.

There is mild fullness of the ventricular system. There is mild prominence of the cerebral cortical sulci and cerebellar folia bilaterally.

A hyperintense signal is seen within the intracranial left internal carotid artery on the T2 Weighted images.

The basal cisternal spaces are unremarkable. There is no shift of the midline structures.


The left internal carotid artery is not visualized from its origin over its entire length. The left middle cerebral artery and its branches are attenuated.

The petrous, cavernous and supraclinoid segments of the right internal carotid artery show normal signal and calibre. The visualized anterior cerebral, right middle cerebral, basilar, vertebral and posterior cerebral arteries also show normal signal, calibre and wall margins. No obvious aneurysm or vascular malformation is identified.

The common carotid arteries and their extracranial branches appear normal bilaterally. There are no vessel wall irregularities or stenosis of the vessels noted.


The MRA features are suggestive of :

1. A haemorrhagic infarct within the left fronto-temporal lobe.

2. Non-visualization of the left internal carotid artery from its origin over its entire length with attenuation of the left middle cerebral artery and its branches.

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