Date : 00.00.00
Name of the Patient : Abc Xyzma R. Tonwalmn / F / 6 yrs.
Referred by : Dr. Abc Xyzah.
Examination : M.R.I. of the Brain.
CLINICAL PROFILE :
H/O ? drop attacks since 0000.
M.R.I of the brain was performed using the following parameters :
5 mm thick T1 Weighted, proton and T2 Weighted axial images.
4 mm thick FLAIR coronal images.
3 mm thick T2 Weighted coronal images.
After administration of contrast, the following parameters were used :
5 mm thick T1 Weighted axial and coronal images with magnetization transfer.
5 mm thick T1 Weighted sagittal images.
There are multiple, small, well marginated, CSF intensity lesions on all the pulse sequences in the subcortical white matter in the frontal and parietal lobes bilaterally and in the genu of the corpus callosum on the right. These lesions vary in size from a few mms to 1.0 cm in diameter. There is no perilesional edema. After administration of contrast, there is no enhancement of these lesions.
The hippocampal complex is unremarkable on either side.
Both the lateral, third and the fourth ventricles are normal. The basal cisternal spaces are unremarkable. There is no shift of the midline structures. No obvious vascular anomaly is identified on this study.
There is no other focal area of abnormal enhancement in the brain parenchyma or the meninges.
Multiple, small, non-enhancing CSF intensity lesions on all the pulse sequences in the subcortical white matter in the frontal and parietal regions bilaterally and in the genu of the corpus callosum as described are not specific for a single etiology. These lesions may represent prominent perivascular spaces or lacunes. Mucopolysaccharidosis should be ruled out.