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Name of the Patient : Abc Xyz Ralmn / F / 64 yrs.
Referred by : Dr. Abc Xyzagwati.
Examination : M.R.I. of the Brain.

CLINICAL PROFILE :

C/O generalized weakness with fluctuating BP and giddiness since 15 days.
Known hypertensive/diabetic.

EXAMINATION :

M.R.I of the brain was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.

5 mm thick FLAIR and Fast Scan (T2 *) coronal images.

5 mm thick T1 Weighted sagittal images.

OBSERVATION :

There is an ill-defined, hypointense lesion on the T1 Weighted images in the left occipital lesion and in the left thalamus. This lesion appears hyperintense on the proton, T2 Weighted and Flair images and most likely represents a recent ischemic lesion. Subtle, hyperintense signal on the T1 Weighted images is noted within the above described lesion in some places which may represent petechial hemorrhage. There is resultant effacement of the sulcal spaces in the left occipital lobe and minimal indentation on the atrium of the left lateral ventricle.

Lacunar infarcts are noted in the cerebellar hemispheres bilaterally (right more than left) and in the left lentiform nucleus.







Ill-defined, hyperintense signal on the proton, T2 Weighted and Flair images are seen in the periventricular white matter bilaterally and in the subcortical white matter in the temporal regions bilaterally which most likely represents ischemic lesions.

There is mild dilatation of both the lateral, third and the fourth ventricles. There is prominence of the cerebral cortical sulci, cerebellar folia and the basal cisternal spaces bilaterally. There is no shift of the midline structures. No obvious vascular anomaly is identified on this study.

IMPRESSION :

1. Altered signal in the left occipital lobe and left thalamus represents a recent ischemic lesion. Petechial haemorrhage is noted in the left occipital lobe

2. Lacunar infarcts in the cerebellar hemispheres bilaterally (right more than left) and in the left lentiform nucleus.

3. Altered signal in the periventricular white matter bilaterally and in the subcortical white matter in the temporal regions bilaterally most likely represents ischemic lesions.

4. Mild cerebral and cerebellar atrophy.