ke/sb/nl/nl
Date : 00.00.00
Name of the Patient : Abc Xyzn Glmn / M / 70 yrs.
Referred by : Dr. Abc Xyzrani.
Examination : M.R.I. of the Brain.
CLINICAL PROFILE :
C/O sudden onset of loss of vision bilaterally.
Known hypertensive.
EXAMINATION :
M.R.I of the brain was performed using the following parameters :
5 mm thick T1 Weighted, proton and T2 Weighted axial images.
5 mm thick FLAIR coronal images.
3 mm thick STIR coronal images.
5 mm thick T1 Weighted sagittal images.
OBSERVATION :
There is an ill-defined, hypointense area in the right occipital lobe on the T1 Weighted images which is seen to follow CSF signal characteristics on all the pulse sequences. Hyperintense areas at the periphery of this lesion on the proton, T2 Weighted and Flair images are hypointense on the T1 Weighted images and would represent areas of gliosis. There is ex-vacuo dilatation of the right lateral ventricle. This lesion would represent an area of cystic encephalomalacia.
Ill-defined hyperintense areas on the proton, T2 Weighted and Flair images are seen in the periatrial white matter and bilateral corona radiata and centrum semiovale. These are hypointense to normal white matter on the T1 Weighted images and are ischemic in etiology.
Lacunar infarcts which are isointense to CSF on all the pulse sequences are seen in the cerebellar hemispheres bilaterally, left side of the pons, in the left thalamus and bilateral lentiform nucleii.
There is slight prominence of the cerebral cortical sulci.
The left lateral, third and the fourth ventricles are normal. The basal cisternal spaces are unremarkable. There is no shift of the midline structures. No obvious vascular anomaly is identified on this study.
Inflammatory changes are seen within the ethmoidal air cells and frontal sinus bilaterally.
The optic nerves show normal signal bilaterally.
IMPRESSION :
1. An area of cystic encephalomalacia in the right occipital lobe.
2. Areas of altered signal in the periatrial white matter and bilateral corona radiata and centrum semiovale are ischemic in etiology.
3. Lacunar infarcts in the cerebellar hemispheres, bilaterally left side of the pons, in the left thalamus and bilateral lentiform nucleii.