Date : 00.00.00
Name of the Patient : Abc Xyzal Abdlmn / M / 7 mnths.
Referred by : Dr. Abc Xyzrani.
Examination : M.R.I. of the Brain.
CLINICAL PROFILE :
H/O visual impairment and does not follow light well.
M.R.I of the brain was performed using the following parameters :
5 mm thick T1 Weighted, proton and T2 Weighted axial images.
4 mm thick T1 Weighted and T2 Weighted coronal images.
There are ill-defined, hypointense areas on the T1 Weighted images in the left temporo-parieto-occipital lobe involving the cortex and subcortical white matter. This lesion follows CSF signal intensity characteristics on all the pulse sequences and represents an area of cystic encephalomalacia. Perilesional white matter hyperintense signal on all the pulse sequences may represent gliotic changes.
Similar changes but to a much lesser extent are noted in the right parieto-occipital region and in the right fronto-parietal cortex.
There is seen a loculated, subdural collection over the left cerebral convexity and posterior interhemispheric fissure. This lesion is slightly hyperintense to CSF on all the pulse sequences. There is no resultant underlying mass effect.
There is mild dilatation of the ventricular system. There is slight thinning of the corpus callosum. There is prominence of the cerebral cortical sulci and the basal cisternal spaces bilaterally. There is no shift of the midline structures. No obvious vascular anomaly is identified on this study.
Inflammatory changes are noted in the left middle ear cavity.
1. Altered signal in the left temporo-parieto-occipital lobes, right parieto-occipital region and in the right fronto-parietal cortex represent areas of cystic encephalomalacia.
2. A loculated, chronic subdural collection over the left cerebral convexity and posterior interhemispheric fissure, without mass effect.
3. Cerebral cortical atrophy with mild ventricular dilatation.
As compared to the previous MRI dated 00.00.00, the left subdural hematoma now appears chronic. Cystic encephalomalacic changes are noted in the brain parenchyma as described. Mild cerebral cortical atrophy with mild ventricular dilatation is now noted.