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sb/ke/rg.

Name of the Patient : Abc Xyzm Milmn / M / 38 yrs.
Referred by : Dr. Abc Xyzrani.
Examination : M.R.I. of the Brain.

CLINICAL PROFILE :

C/O left hemiparesis since 00.00.0000.
H/O similar complaints in October 0000.
Known hypertensive.

EXAMINATION :

M.R.I of the brain was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.

5 mm thick FLAIR coronal images.

OBSERVATION :

There is an ill-defined, hyperintense signal on the proton, T2 Weighted and FLAIR images in the right high parietal parafalcine cortex and along the right fronto-parietal cortex. This lesion appears hypointense to normal white matter on the T1 Weighted images and represents an ischemic lesion, most likely recent, in the given clinical setting. Subtle, hyperintense signal on T1 Weighted images is noted within this lesion which may represent hemoglobin breakdown products. Evidence of haemorrhage is noted within this lesion.

There is a CSF intensity lesion on all pulse sequences in the right posterior parietal, parafalcine cortex. Perilesional white matter hyperintense signal on the T2 Weighted and FLAIR images may represent gliotic changes.

There is head of caudate nulceuis on the left side.

A lacunar infarct is noted in the left frontal deep white matter.

Both the lateral, third and the fourth ventricles are normal. The basal cisternal spaces are unremarkable. There is no shift of the midline structures. No obvious vascular anomaly is identified on this study.
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Inflammatory changes are noted in the right maxillary antrum.

IMPRESSION :

1. Altered signal in the right high parietal parafalcine cortex and along the right fronto-parietal cortex represents an ischemic lesion, most likely recent, in the given clinical setting.

2. Altered signal in the right posterior parietal, parafalcine cortex represents encephalomalacic changes, most likely the sequelae of a previous vascular insult.

3. A lacunar infarct in the left frontal deep white matter.


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